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Abstract Details

Evaluation Of Stroke Etiology With Post-thrombectomy Tcd Vessel Velocities
Cerebrovascular Disease and Interventional Neurology
Cerebrovascular Disease and Interventional Neurology Posters (7:00 AM-5:00 PM)
120

To assess whether stroke etiology can be evaluated from post-thrombectomy TCD vessel velocities

 

Two major entities accounting for large vessel occlusion strokes are embolism from a proximal source and intracranial atherosclerotic disease (ICAD). Thrombolysis in cerebral infarction (TICI) scores are used to grade post-endovascular revascularization (0 – no reperfusion, 1-2A – partial reperfusion, 2B-3 – adequate reperfusion). Transcranial dopplers (TCD) provide information regarding post-thrombectomy vessel velocities, but have not been utilized in evaluating stroke etiologies. We sought to assess whether the stroke etiology can be further defined by using TCD data of post-thrombectomy vessels.
We retrospectively reviewed TCD velocities of patients who underwent thrombectomy within 10 days. Clinical characteristics and demographics were reviewed. Patients with ICAD and embolic strokes were identified, and post thrombectomy target vessel velocities were compared between the two etiologies. The data was further stratified by TICI score and velocities were compared within the same TICI score group (0-2A, 2B-3).
Fifty patients were analyzed (mean age 61.5, 60% male, 17 black, 30 white, 1 Asian, 2 unknown). 31 patients had embolic strokes, 5 ICAD, 11 unknown (or two compelling etiologies) and 3 other etiologies. In patients with TICI 2B-3 reperfusion, 20 patients with embolic stroke had a mean velocity of 55.4 +/- 16.7 cm/s, 3 patients with known ICAD had 44.3 +/- 22.9 cm/s, 8 patients with unknown etiology had 57.1 +/- 37.0 cm/s, and 2 others had 62.5 +/- 5.3 cm/s (P- value 0.82). There was no significant difference in post-thrombectomy mean velocities between embolic and ICAD strokes.
There were no significant velocity differences between post-thrombectomy target vessels, grouped by TICI score and different etiologies of stroke. TCD target velocity alone is not sufficient to differentiate between embolic and ICAD etiologies.
Authors/Disclosures
Khine Yadanar Sein, MD
PRESENTER
Dr. Sein has nothing to disclose.
Hely Nanavati, MBBS (University of Alabama At Birmingham) Hely Nanavati has received personal compensation for serving as an employee of University of Alabama At Birmingham.
Soo Young Kwon, MD (Stanford University) Dr. Kwon has nothing to disclose.
Chen Lin, MD (LSU Health Shreveport) The institution of Dr. Lin has received research support from VA.