Stroke is the main cause of epilepsy in adults. PSE is defined as the presence of two or more unprovoked seizures 7 days after a stroke or a single unprovoked seizure 30 days after. 

Several risk factors for the development of PSE have been described, such as cortical envolvement, anterior circulation stroke, intraparenchymal hemorrhage amongst others.

We retrospectively reviewed medical records from patients admitted to our hospital with diagnosis of stroke between 2016-2018, we excluded those who died within the first week. Statistical analysis was performed with SPSS analysing ischemic stroke with and without hemorrhagic transformation, hemorrhagic stroke, localization, vascular territory and number of brain lobes envolved, acute symptomatic seizure (ASS) and epilepsy development.

251 patients were included, 14.3% developed epilepsy (36), 8% had ASS (21) from which 14 developed PSE (p:0,001). Mean age was 71 (32-95). From those who developed PSE 38.8% were women (14), 61.2 men (22). 76% had ischemic stroke, 24% hemorrhagic. The following were associated with PSE: ischemic stroke (p: 0.012), hemorrhagic stroke (p: 0.005), parietal (p: 0.007), temporal (p: 0.001), corticosubcortical (p: 0,001), evolvement of two lobes (p: 0.011).  Patients with cerebellar and/or subcortical stroke did not develop PSE (p: 0.035 y p: 0.003 respectively). The prevalence of PSE was 14.3%, the anual incidence was 7%.

Prevalence and incidence in our population was similar to the reported in the literature. We found statistical significance regarding the development of PSE with ischemic and hemorrhagic stroke, parietal, temporal, corticosubcortical and bilobar envolvement. These do not differ from the risk factors described in the literature. In our population subcortical or cerebellar stroke did not develop PSE.